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One of the hallmarks of aging is a decline in energy metabolism in many tissues, especially liver, heart, and skeletal muscle

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Dysfunction of ETC has been associated with various neurological diseases from infantile-onset neurodevelopmental regression (such as Leigh syndrome) to adult-onset optic neuropathy or peripheral neuropathy (such as Leber hereditary optic neuropathy (LHON) in cI deficiency, and neurogenic muscle weakness, ataxia, and retinitis pigmentosa (NARP) in cV deficiency) (Gorman et al., 2016)