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Similar analyses were performed for the risk of Alzheimer disease (subjects in whom other types of dementia developed during follow-up were censored at the date of the diagnosis of dementia)
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Recent studies have validated this general approach demonstrating that molecules designed around angiotensin IV (Yamamoto et al, 2010) or the hinge sequence itself (Kawas et al., 2011) can bind HGF, block its dimerization, and attenuate HGF-dependent cellular actions
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